Quan Tran

Education and training

Stanford

Research

Publications

21 attributed works. Counts reflect resolved authorship and may be incomplete.

• 2026

  Effect of race and sex on lupus diagnosis in primary care: A randomized factorial survey study.

  PloS one0 citesPMID 41650210
• 2026

  CD39+CD49a+CD103+ cytotoxic tissue-resident natural killer cells infiltrate and control solid epithelial tumor growth in mice

  Science Translational Medicine
• 2025

  Supplementary Figure 7 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Figure 4 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Figure 1 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Data from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Figure 6 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Figure 5 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Figure 2 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage

  Cancer Discovery
• 2025

  Supplementary Figure 8 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Table 2 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Figure 9 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Figure 3 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Dataset from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2025

  Supplementary Table 1 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
• 2024

  TeleHelp Ukraine: A distributed international telemedicine response to the ongoing war.

  Journal of global healthRCR 0.964 citesPMID 39451063
• 2023

  Oral cavity cancer in young, non-smoking, and non-drinking patients: a contemporary review.

  Critical reviews in oncology/hematologyRCR 7.5150 citesPMID 37633348
• 2021

  Humanized Mouse Models for the Advancement of Innate Lymphoid Cell-Based Cancer Immunotherapies

  Frontiers in Immunology
• 2021

  Targeting KRAS4A splicing through the RBM39/DCAF15 pathway inhibits cancer stem cells

  Nature Communications

Sources

Roster sources establish the listed training role. Additional education, career, and research sources are added as the profile is enriched.

• Internal Medicine Residency roster

Program context: Internal Medicine Residency